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HAIR LOSS (BALDING)
ABSTRACTS
Page 1

Balding hair follicle dermal papilla cells contain higher levels of androgen receptors than those from non- balding scalp.
Hibberts NA, Howell AE, Randall VA
Department of Biomedical Sciences, University of Bradford, UK.
J Endocrinol (England) Jan 1998, 156 (1) p59-65

Androgens can gradually transform large scalp hair follicles to smaller vellus ones, causing balding. The mechanisms involved are unclear, although androgens are believed to act on the epithelial hair follicle via the mesenchyme-derived dermal papilla. This study investigates whether the levels and type of androgen receptors in primary lines of cultured dermal papilla cells derived from balding scalp hair follicles differ from those of follicles from non-balding scalp. Androgen receptor content was measured by saturation analysis using the non-metabolisable androgen, [3H]mibolerone (0.05-10 nM) in a 9-10 point assay. Pubic dermal fibroblasts and Shionogi cells were examined as positive controls. Repetitive assays of Shionogi cells showed good precision in the levels of androgen receptor content (coefficient of variation = 3.7%). Specific, high affinity, low capacity androgen receptors were detected in dermal papilla cells from both balding and non-balding follicles. Balding cells contained significantly (P < 0.01) greater levels of androgen receptors (Bmax = 0.06 +/- 0.01 fmol/10(4) cells (mean +/- S.E.M.)) than those from non-balding scalp (0.04 +/- 0.001). Competition studies with a range of steroids showed no differences in receptor binding specificity in the two cell types. The higher levels of androgen receptors in cells from balding scalp hair follicles with similar properties to those from non-balding scalp concur with the expectations from their in vivo responses to androgens. This supports the hypothesis that androgens act via the dermal papilla and suggests that cultured dermal papilla cells may offer a model system for studying androgen action in androgenetic alopecia.

United States Patent
Peter H. Proctor
4126 Southwest Freeway, Suite 1616,
Houston, Tex. 77027

United States Patent [19][11]
Patent Number: 5,352,442
Proctor [45]
Date of Patent: Oct. 4,1994

[54] TOPICAL TEMPO

[76] Inventor: Peter H. Proctor,
4126 Southwest Freeway,
Suite 1616, Houston, Tex. 77027

[21] Appl. No.: 21,970
[22] Filed: Feb. 24, 1993

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OTHER PUBLICATIONS

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Ando et al., Chemical Abstracts 93:79872n (1980).

Bazzano et al., Journal of American Academy of Dermatology,
vol. 15, pp. 880-883 (1986).

Barry, Pharmacology of the Skin,
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Cheng et al., Archives of Dermatological Research,
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Cumming. et al., Journal of American Medical Association,
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Dawber, Dermatologica,
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DeVillez, Archives of Dermatology,
vol 121, pp. 197-202, (1985)

Dostert a d., Xenobiotica,
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Ehman et d., Investigative Radiology,
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Feelisch et al., Evr. Journal of Pharmacology,
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Feelisch et al., Evr. Journal of Pharmacology,
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Fox et al., Annals of the New York Academy of Sciences,
vol. 411, pp. 14-19 (1983).

Primary Examiner-Jose G. Dees
Assistant Examiner-Porfirio Nazario-Gonzalez
Attorney, Agent or Firm-Daniel N. Lundeen; Andrew S. Pryzant

[57] ABSTRACT

Topical 2,2,6,6-tetramethyl-1-piperdinyloxyl (TEMPO) is disclosed. The compound has utility in a topical pharmaceutical formulation for the cosmetic treatment of hair loss and the cosmetic stimulation of hair growth.

10 claims, No Drawings
United States Patent [19]

Proctor US005472687A [11] Patent Number: 5,472,687
[45] Date of Patent: Dec. 5,1995
[54] TOPICAL PYRIDINE N-OXIDES
[76] Inventor: Peter H. Proctor, Twelve Oaks
Medical Towers 4126 SW. Freeway,
Suite 1616, Houston, Tex. 77027

[21] Appl. No.: 193 ,228 [22] Filed: Feb. 7, 1994

Bourayne et al., Journal de chirnie physique,
vol. 79, No. 2, pp. 139-147 (1982).

Anderson, Chemical Abstracts,
vol. 90, pp. 311K (1979).

Ando et al., Chemical Abstracts,
93:79872n (1980).

Bazzano et al., Journal of American Academy of Dermatology
vol. 15, pp. 880-883 (1986).

Berry, Pharmacology of the Skin,
vol. 1, pp. 121-137 (1987).

Cheng et al., Archives of Dermatological Research,
vol. 278, pp. 470-473 (1986).

Cumming et al., Journal of American Medical Association,
vol. 247, pp. 1295-1298 (1982).

Current Therapy,
pp. 599-603 (1984).

Dahl, Men's Fitness,
pp. 93-95 (Feb. 1989).

Dawber, Dermatalogical
vol. 175, suppl. 2, pp. 23-28 (1987).

DeVillez, Archives of Dermatology
vol. 121,pp. 197-202, 1985. Dermatologica,
vol. 175 suppl. 2, pp. 1-56 (Oct. 87).

Dostert et al., Xenobiotica,
vol. 15, No. 10, pp. 799-803 (1985).

Ehman et al., Investigative Radiology,
vol. 21, pp. 125-131 (1986).

Feelisch et al., Evr. Journal of Pharmacology,
vol 139, pp. 19-30 (1987).

Feelisch et al., Evr Journal of Pharmacology,
vol. 142, pp. 405-409 (1987).

Fiedler, Dermatologica,
vol. 175, suppl. 2, pp. 29-35 (1987).

Fox et al., Annals of the New York Academy of Sciences,
vol. 411, pp. 14-19 (1983).

Goffman et al., International Journal of Radiation, Oncology, Biology and Physics,
vol. 22, pp. 803-806 (Nov. 4, 1992).

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Primary Examiner-Porfirio Nazario-Gonzales Attorney, Agent, or Firm-Sroufe, Payne & Lundeen

ABSTRACT

Topical pyridine N-oxides, including 3-carboxypyridine N-oxide, niacinamide N-oxide, and a-(4-pyridyl-1-oxide)N-t-butylnitrone, are disclosed. The topical compositions have utility in a topical pharmaceutical formulation for the cosmetic treatment of hair loss and the cosmetic stimulation of hair growth.

10 Claims, No Drawings

OTHER PUBLICATIONS

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vol. 89, No. 1, pp. 98-102 (1979).

Dooley et al., Biochemical and Biophysical Research Communications,
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Jahagirdar et al., Chemical Abstracts,
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Straehle et al., Chemical Abstracts,
vol. 93, No. 3, Chem Abs. No. 21526m (1980).

Anderson, Chemical Abstracts,
vol. 90, pp. 311K (1979): Ando et al., Chemical Abstracts, 93:79872n (1980).

Bazzano et al., Journal of American Academy of Dermatology,
vol. 15, pp. 880-883 (1986).

Berry, Pharmacology of the Skin,
vol. 1, pp. 121-137 (1987).

Cheng et al., Archives of Dermatological Research,
vol. 278, pp. 470-473 (1986).

Cumming et al., Journal of American Medical Association,
voL 247, pp. 1295-1298 (1982).

Current Therapy,
pp. 599-603 (1984).

Dahl, Men's Fitness,
pp. 93-95 (Feb. 1989).

Dawber, Dermatologica,
vol. 175, suppl. 2, pp. 23-28 (1987).

DeVillez, Archives of Dcrmatology,
vol. 121, pp. 197-202, (1985).

Dermatologica,
vol. 175, suppl. 2, pp. 1-56 (Oct. 1987).

Dostert et al., Xenobiotica,
vol. 15, No. 10, pp. 799-803 (1985).

Ehman et al., investigative Radiology,
vol. 21, pp. 125-131 (1986).

Feelisch et al., Evr. Journal of Pharmacology,
vol. 139, pp. 19-30 (1987).

Feelisch et al., Evr. Journal of Pharmacology,
vol. 142, pp. 405-409 (1987).

Fiedler, Dermatologica,
vol. 175, suppl. 2, pp. 29-35 (1987).

Pox et al., Annals of the New York Academy of Sciences,
vol. 411, pp. 14-19 (1983).

Goffman et al., International Journal of Radiation, Oncology, Biology and Physics,
vol. 22, pp. 803-806 (Nov. 4, 1992).

Headington, Current Therapeutic Research,
vol. 36, pp. 1098-1105 (1984).

Hearse ct al., Circulation Research,
vol. 60, pp. 375-383 (1987).

Primary Examiner-Porfirio Nazario-Gonzales Attorncy, Agent, or Firm-Sroufe, Payne & Lundeen

[57] ABSTRACT

SOD for treating hair loss is disclosed. The SOD has utility in a topical pharmaceutical formulation for the cosmetic treatment of hair loss and the cosmetic stimulation of hair growth. The SOD comprises copper salicylate, copper aspirinate, indomethacin-copper, or a complex of an amino acid or peptide and a transition metal.

19 Claims, No Drawings


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